🧠 Dementia: Biomarkers & Imaging

เปรียบเทียบ 8 ประเภท — Biomarkers (AT(N)) + Imaging

A

Amyloid
CSF Aβ42 ↓ · Amyloid PET+

T

Tau
CSF p-tau ↑ · Tau PET+

N

Neurodegeneration
CSF t-tau ↑ · FDG-PET · MRI

Alzheimer's Disease
Most common cause of dementia (60-70%)
Aβ42 ↓or Aβ42/40 ratio ↓
p-tau 181/217 ↑most specific
t-tau ↑neurodegeneration
p-tau 217NfL ↑
MRI:Hippocampal & entorhinal atrophy
FDG-PET:Temporoparietal hypometabolism
Amyloid PET:+
Tau PET:+
Aβ plaques + NFT (Braak I-VI) → neuronal loss
Lewy Body Dementia
α-synuclein (Lewy bodies) — 2nd most common
Fluctuating cognitionVisual hallucinationsParkinsonismRBD
RT-QuIC:α-synuclein seed detection
NfL ↑may be higher vs AD
MRI:Relatively preserved hippocampus
FDG-PET:Occipital hypometabolism
DAT-SPECT:− Loss of DAT
MIBG:− Reduced cardiac uptake
Parkinson's Disease Dementia
α-synuclein — same pathology as DLB
Parkinsonismpreceded dementia by ≥1 year
Same as DLBα-synuclein RT-QuIC +
DAT-SPECT:− Loss of dopaminergic transporters
FDG-PET:Posterior cortical hypometabolism
MRI:Often normal early
Frontotemporal Dementia
bvFTD · svPPA · nfvPPA — Tau or TDP-43
bvFTDSemantic PPAnfvPPA
Tau FTD:p-tau ↑ (Pick's disease)
TDP-43 FTD:TDP-43 in CSF (50%)
NfL ↑all subtypes
GRN mutation:progranulin ↓
MRI:"Knife-edge" frontal/temporal atrophy
FDG-PET:Frontal/temporal hypometabolism
Tau PET:− (vs AD)
Vascular Dementia
Cerebrovascular disease — 2nd most common
Aβ42:Normal
t-tau:Normal or mildly ↑
NfL ↑vascular injury
MRI:Multi-infarct pattern, lacunes
WMH:White matter hyperintensities (Fazekas)
Microbleeds:Caused by amyloid or hypertension
DAT-SPECT:+ Normal
Stepwise decline+ focal neurological signs
Normal Pressure Hydrocephalus
🍷 Potentially REVERSIBLE — "Wet, Wacky, Wobbly"
Urinary incontinenceDementiaGait apraxia
No specific biomarker
CSF pressure:Normal (hence "normal pressure")
Evans index:>0.3 (frontal horn / skull)
Ventricular enlargementdisproportionate to atrophy
Callosal angle:<90° (upward deviation)
MRI:Transependymal CSF egress
💡VP shunt can reverse symptoms — always consider!
Creutzfeldt-Jakob Disease
⚡ Prion disease — FASTEST progression (weeks-months)
14-3-3 protein:Neuronal destruction (↑)
Tau:>1000 pg/mL (very high!)
RT-QuIC:Prion seed detection
NfL:Extremely high (rapid neurodegeneration)
MRI DWI:Cortical ribboning + basal ganglia hyperintensity
FDG-PET:Diffuse cortical hypometabolism
EEG:Periodic sharp waves (1-2 Hz)
⚠️Rapid progression: death usually <1 year from onset
Wernicke-Korsakoff Syndrome
🍷 Thiamine (B1) deficiency — usually chronic alcohol
AtaxiaOphthalmoplegiaConfusion
Anterograde amnesia+ confabulation
Blood thiamine ↓
CSF thiamine ↓
MRI:Mammillary body atrophy, medial thalamic T2 hyperintensity, periaqueductal gray
CT:Ventricular enlargement possible
Wernicke: acute, reversible with B1. Korsakoff: often permanent.

📊 Quick Reference Table

Type Key Biomarker Key Imaging Distinguishing Feature AT(N)
🔵 Alzheimer's Aβ42 ↓, p-tau 217 ↑ Hippocampal atrophy, Amyloid PET+ Memory loss, amyloid/tau PET both + A+T+N+
🩷 Lewy Body α-synuclein RT-QuIC + DAT-SPECT −, Occipital hypometabolism Fluctuating cognition, visual hallucinations A−T−N−
🟣 PDD Same as DLB DAT-SPECT − Parkinsonism ≥1yr before dementia A−T−N−
🟪 FTD p-tau ↑ or TDP-43, NfL ↑ Frontal/temporal atrophy, Tau PET − Personality change, language deficits A−T−N+
🟠 Vascular Aβ normal, NfL ↑ Multi-infarct, WMH, DAT-SPECT + Stepwise decline, focal signs A−T−N+
🟡 NPH None specific Evans index >0.3, Ventricular ↑ Gait first → reversible with VP shunt! A−T−N−
🔴 CJD 14-3-3 ↑, Tau >1000, RT-QuIC + Cortical ribboning (DWI) Rapid progression (weeks-months) A−T−N+
🟢 Wernicke-K Thiamine ↓ Mammillary body atrophy (MRI) Ataxia + ophthalmoplegia + confusion A−T−N−
Dementia Biomarkers & Imaging Comparison · AT(N) Framework (NIA-AA 2024)